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Aspirin suppresses prostaglandin (which inhibits platelet activation and dilates blood vessels) and thromboxane (which encourages platelet activation and constricts vessels).
Prostacyclin stops blood clotting while thromboxane , as the name suggests, induces thrombus formation.
As a platelet is activated it can release ADP and thromboxane , both potent autocatalytic molecules.
The sulfide metabolite of sulfinpyrazone affects platelet aggregation by inhibiting thromboxane production.
Cocaine enhances platelet aggregation as a result of increased levels of platelet agonists thromboxane [A.sub.2] and adrenaline.
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