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Herein we present data on symptoms, alterations in peripheral lymphocyte phenotypes, and autoantibodies observed in adult patients.
No tests for autoantibodies should be performed without a clinical evaluation that leads to a presumptive diagnosis.
Examples of these substances include plasma proteins such as albumin and immunoglobulins, pathogenic autoantibodies , clotting factors, endotoxins, and proinflammatory cytokines.
All patients had significant increases in autoantibodies against neural antigens.
Autoantibodies such as antinuclear antibodies and myositis-specific autoantibodies bind to proteins normally present inside one's own normal skin and muscle cells.
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